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Discussion In this work, we establish the N-terminal tail of GNMT as a key regulatory domain that maintains SAM-binding site stability and coordinates 5mTHF-mediated negative feedback, thereby preserving methyl-donor homeostasis and cellular methylation potential

صن سكرين ابيب

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212−221 With the emergence of cell 3D-printing technologies using pluripotent stem cells or differentiated cells as inks, impressive progress has been successful in the formation of heterogeneous tissues